Hemifacial Spasm in Mucopolysaccharidosis Type VI (Maroteaux–Lamy Syndrome)

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Hemifacial spasm and neurovascular compression syndrome.

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Hemifacial spasm developed in a 47-year-old man with tuberculous meningitis. The spasms ceased completely following vigorous antituberculous treatment. A selective compression of the facial nerve root along its exit at the brainstem by a localised inflammatory process is the most tenable explanation for the development of the hemifacial spasm.

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Botulinum toxin type B in blepharospasm and hemifacial spasm

Botulinum neurotoxins (BTXs) inhibit the presynaptic release of acetylcoline causing a chemical denervation that results in sustained muscle weakness and have been used in the past 20 years to induce selective blocking of hyperactive striatal (and smooth) muscles. All the different seven serotypes of BTXs have in common the mechanism of action (block of the neuroexocytosis machinery inside the ...

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ژورنال

عنوان ژورنال: Tremor and Other Hyperkinetic Movements

سال: 2018

ISSN: 2160-8288

DOI: 10.5334/tohm.413